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Testosterone Therapy Video Presentation HCG Therapy Video Presentation

MALE FERTILITY TREATMENT:
HCG THERAPY TO INCREASE NATURAL
TESTOSTERONE PRODUCTION IN MEN
AND INCREASED SPERM COUNT WHEN
COMBINED WITH EXISTING FSH TO CREATE
SPERMATOGENESIS

National Medical Clinic, Inc. physicians provide Human Chorionic Gonadotropin (HCG) hormone therapy to increase male fertility (sperm count) when the patient has sufficient naturally produced Follicle-Stimulating Hormone (FSH) to enable the administration HCG to potentially increase spermatogenesis in hypogonadotropic men deficient in LH and sperm count.

In males, FSH enhances the production of androgen-binding protein by the Sertoli cells of the testes, and is critical for spermatogenesis. FSH regulates the reproductive processes of the human body. Both LH (or HCG as a medication substitute for naturally produced LH) and FSH must be present for spermatogenesis.

The purpose of HCG treatment with regard to male infertility is to increase spermatogenesis in hypogonadotropic men deficient in LH who have sufficient FSH. If the patient has insufficient FSH, then they should consult with a reproductive endocrinologist who may administer both FSH and HCG. FSH is available mixed with LH in the form of Pergonal or Menopur, and other more purified forms of urinary gonadotropins, as well as in a pure forms as recombinant FSH (Gonal F, Follistim).

Hypogonadism is a medical term for a defect of the reproductive system that results in lack of function of the gonads (ovaries or testes). The gonads have two functions: to produce hormones (testosterone, estradiol, antimullerian hormone, progesterone, inhibin B), activin and to produce gametes (eggs or sperm). Deficiency of sex hormones can result in defective primary or secondary sexual development, or withdrawal effects (e.g., premature menopause) in adults. Defective egg or sperm development results in infertility.

Spermatogenesis is the process by which male spermatogonia develop into mature spermatozoa. Spermatozoa are the mature male gametes in many sexually reproducing organisms.

Spermatogenesis produces mature male gametes, commonly called sperm but specifically known as spermatozoa, which are able to fertilize the counterpart female gamete, the oocyte, during conception to produce a single-celled individual known as a zygote. This is the cornerstone of sexual reproduction and involves the two gametes both contributing half the normal set of chromosomes (haploid) to result in a chromosomally normal (diploid) zygote.

Spermatogenesis takes place within several structures of the male reproductive system. The initial stages occur within the testes and progress to the epididymis where the developing gametes mature and are stored until ejaculation. The seminiferous tubules of the testes are the starting point for the process, where stem cells adjacent to the inner tubule wall divide in a centripetal direction—beginning at the walls and proceeding into the innermost part, or lumen—to produce immature sperm. Maturation occurs in the epididymis and involves the acquisition of a tail and hence motility.

Hormonal control of spermatogenesis varies among species. In humans the mechanism are not completely understood, however it is known that initiation of spermatogenesis occurs at puberty due to the interaction of the hypothalamus, pituitary gland and Leydig cells. If the pituitary gland is removed, spermatogenesis can still be initiated by follicle stimulating hormone and testosterone.

Follicle stimulating hormone stimulates both the production of androgen binding protein by Sertoli cells, and the formation of the blood-testis barrier. Androgen binding protein is essential to concentrating testosterone in levels high enough to initiate and maintain spermatogenesis, which can be 20-50 times higher than the concentration found in blood. Follicle stimulating hormone may initiate the sequestering of testosterone in the testes, but once developed only testosterone is required to maintain spermatogenesis. However, increasing the levels of follicle stimulating hormone will increase the production of spermatozoa by preventing the apoptosis of type A spermatogonia. The hormone inhibin acts to decrease the levels of follicle stimulating hormone.

The Sertoli cells themselves mediate parts of spermatogenesis though hormone production. They are capable of producing the hormones estradiol and inhibin. The Leydig cells are also capable of producing estradiol in addition to their main product testosterone.

In this program HCG is a hormone prescribed for men by NMC Physicians to treat male infertility (low sperm count) by functioning as a substitute for the deficient secretion of LH by the pituitary gland. Similar to naturally produced LH, the HCG administered in this treatment stimulates the testes to increase testosterone production and spermatogenesis or the process of creating sperm. This treatment requires that a patient present with a sufficient level of naturally produced FSH. The gonadotropin FSH in combination with HCG induces spermatogenesis in hypogonadotropic men. This treatment does not include administering both recombinant FSH and LH to induce spermatogenesis.

HCG is approved for use in cases of hypogonadotropic hypogonadism (hypogonadism secondary to a pituitary deficiency). It is used to stimulate the testes of men who are hypogonadal or lack sufficient testosterone production.

Diagnosis of Oligozoospermia

No single medical treatment has proven to be consistently reliable in increasing sperm count for patients diagnosed with Oligozoospermia or low sperm count. A diagnosis of Oligozoospermia is based on a symptom wherein a sample of semen contains less than 20 million spermozoa per ml of ejaculate.

Increasing Spermatogenesis in Hypogonadotropic Men

"Like urinary FSH, recombinant FSH in combination with HCG seems to induce spermatogenesis in hypogonadotropic men" See Drug Treatment of Male Fertility Disorders by Gerhard Haidl, et al. and specifically the discussion of HCG therapy therein. This article is located at URL: http://www.asiaandro.com/1008-682X/2/81.htm

The Decline in Gonadal Stimulating Pituitary Hormone LH (Leutenizing hormone)

The natural decline in male testosterone production that occurs with aging is attributed to a decline in the gonadal stimulating pituitary hormone LH (Leutenizing hormone). As a result of the hypothalamus secreting less gonadoropin-releasing hormone (GhRH), which stimulates the pituitary gland to produce LH, the pituitary gland produces declining amounts of LH. This decrease in the pituitary secretion of LH reduces the stimulation of the gonads or male testes and results in declining testosterone and sperm production due to the decreased function of the gonads.

The decreased stimulation of the testes by the pituitary's diminished secretion of LH can also cause testicular atrophy. HCG stimulates the testis in the same manner as naturally produced. HCG Therapy is administered medically to increase male fertility by stimulating the testes to produce more sperm cells and thereby increase sperm count or Spermatogenesis.

How HCG Therapy Increases Plasma Testosterone Level in Hypogonadotropic Men

HCG therapy uses the body's own biochemical stimulating mechanisms to increase plasma testosterone level during HCG therapy. It is used to stimulate the testes of men who are hypogonadal or lack sufficient testosterone.

The male endocrine system is responsible for causing the testes to produce testosterone. The HPTA (hypothalamic-pituitary-testicular axis) regulates the level of testosterone in the bloodstream. and . The hypothalamus produces gonadotropin-releasing hormone (GnRH), which stimulates the pituitary gland to release Leutenizing hormone (LH).

LH released by the pituitary gland then travels from the pituitary via the blood stream to the testes where it triggers the production and release of testosterone. Without the continuing release of LH by the pituitary gland, the testes would shut down their production of testosterone, causing testicular atrophy and stopping natural testosterone produced by the testes.

As men age the volume of hypothalamus produced gonadotropin-releasing hormone (GnRH) declines and causes the pituitary gland to release less Leutenizing hormone (LH). The reduction if the volume of LH released by the Pituitary gland decreases the available LH in the blood stream to stimulate the testes to produce testosterone.

In males, HCG mimics LH and increases testosterone production in the testes. As such, HCG is administered to patients to increase endogenous (natural) testosterone production. The HCG medication administered combines with the patient's own naturally available LH released into the blood stream by the Pituitary gland and thereby increases the stimulation of the testes to produce more testosterone than that produced by the Pituitary released LH alone. The additional HCG added to the blood stream combined with the Pituitary gland's naturally produced LH triggers a greater volume of testosterone production by the testes, since HCG mimics LH and adds to the total stimulation of the testes.

In this treatment HCG is administered to men to promote an increase in sperm production (spermatogenesis) by the testes. HCG combined FSH or both LH and FSH is also used to increase male spermatogenesis and medically treat male infertility. (Source: Wikipedia)

Patients May Incur Additional Medical Laboratory Diagnostic Testing Fees

In addition to our published fees for this treatment, the patient shall incur additional costs for sperm count or male fertility testing. The patient can secure requested male fertility diagnostic tests through their primary physician and such tests are covered by medical insurance coverage accepted by the patient's primary physician. The effective out-of-pocket costs for such testing can be reduced thereby.

A High Protein Diet Combined with Nutritional Supplements Consisting of Amino Acids, Vitamin C and Anti-oxidants is Recommended During the Treatment Period to Repair Sperm Cell DNA damage

During this medical treatment , it is requested that you follow a high protein diet combined with amino acids vitamin E, Vitamin C and anti-oxidants to reduce DNA damage in the sperm cells that are produced during the course of treatment.

Conditional Enrollment and Refund of Patient's Medical Retainer Fee Paid Clinic

A patient's enrollment in a medical treatment program is subject to the approval of the assigned National Medical Clinic, Inc treating physician. The approval of a treatment program and issuance of written prescriptions is based upon the patient's medical complaint, symptoms (subjective findings), medical laboratory diagnostic testing (objective findings), patient's physical examination results, patient's medical history, physician's clinical assessment-evaluation of the patient and there existing a nexus relationship between the medical condition to be treated and the mediation prescribed. If the treating physician declines to prescribe the patient's sought treatment, then National Medical Clinic, Inc. shall returns to the patient 100% of the retainer fee/purchase funds paid less the incurred fees for the physical examination and clinical assessment of the patient, laboratory diagnostic blood testing and clinic fees.

While there are no guarantees in the practice of medicine, we look forward optimistically to working with you to jointly achieve a successful result for both you and your spouse or female partner.

Other Medical Treatments to Increase Male Fertility

Your response to the above male medical fertility treatment, as well as, male infertility medical laboratory diagnostic test reports may indicate to your treating physician that a different male medical fertility treatment is indicated to enhance the probability of your achieving increased male fertility. If the treating physician elects not to provide this new treatment, they may refer you to another physician or clinic for further evaluation and treatment.

National Medical Clinic, Inc. Male Medical Fertility Treatments include:
Medical Treatments for Male Patients with Insufficient LH to Increase Testosterone Production by Testes:

  • Pharmacy Compounded or Pharmaceutically Manufactured HCG to Increase Testosterone Production
  • Pharmaceutically Manufactured Urinary LH to Increase Testosterone Production
  • HCG and Pharmaceutically Manufactured Urinary LH to Increase Testosterone Production
Medical Treatments for Male Patients with Insufficient FSH Production to Increase Available FSH:
  • Pharmaceutically Manufactured Urinary FSH
  • Pharmaceutically Manufactured Recombinant FSH (Gonal F, Follistim)
Medical Treatments for Male Patients with Insufficient LH and FSH Production to
Increase Testosterone Production by Testes and Available FSH:
  • Combined HCG and Pharmaceutically Manufactured Urinary FSH
  • Combined Pharmaceutically Manufactured Urinary LH and FSH (Pergonal or Menopur)
  • Combined HCG, Pharmaceutically Manufactured Urinary LH and FSH (Pergonal or Menopur)
  • Combined Pharmaceutically Manufactured Urinary LH and Recombinant FSH (Gonal F, Follistim)
  • HCG, Combined Pharmaceutically Manufactured Urinary LH and Recombinant FSH (Gonal F, Follistim)

"Like urinary FSH, recombinant FSH in combination with HCG seems to induce spermatogenesis in hypogonadotropic men" see Drug Treatment of Male Fertility Disorders by Gerhard Haidl, et al. and specifically the discussion of HCG therapy therein. This article is located at URL: http://www.asiaandro.com/1008-682X/2/81.htm

More Information Regarding the Use of Medications to Treat Male Infertility

Information about male fertility treatments is available at the National Medical Clinic, Inc. website
www.nationalmedicalclinic.com
Information about male fertility treatments is also available at URL: http://en.wikipedia.org/wiki/Human_Chorionic_Gonadotropin#Use_as_medication – use of HCG to increase fertility in men and women and http://content.nejm.org/cgi/content/extract/332/5/312 - Treatment of Male Infertility

ART or Assisted Reproductive Technology

If the HCG therapy, the FSH mixed with LH in the form of Pergonal or Menopur treatment and recombinant FSH (Gonal F, Follistim) and LH treatment do not increase sperm count sufficiently, then you may want to consider use of ART or assisted reproductive technology. National Medical Clinic, Inc. does not offer Assisted Reproductive Technology to treat infertility.

Clinical Pharmacology - HCG:

The action of HCG is virtually identical to that of pituitary LH, although HCG appears to have a small degree of FSH activity as well. It stimulates production of gonadal steroid hormones by stimulating the interstitial cells (Leydig cells) of the testis to produce androgens.

HCG is compounded or manufactured in various quantities, including 5,000 to 20,000 IU (International Units) per 10 cc vials.

HCG is a natural protein hormone secreted by the human placenta and purified form the urine of pregnant women. This hormone is not a natural male hormone but mimics the natural hormone LH (Leutenizing Hormone) almost identically. This LH stimulates the production of testosterone by the testis in males.

Thus HCG sends the same message and results in increased testosterone production by the testis due to HCG's effect on the leydig cells of the testis. HCG therapy uses the body's own biochemical stimulating mechanisms to increase plasma testosterone level.

When administered, HCG raises serum testosterone very quickly. A rise in testosterone first appears in about two hours after injecting HCG. The second peak occurs about two to four days later. HCG is not a steroid and is administered to assists the body in the continuing production of its own natural testosterone as a result of LH signals stimulating production of testosterone by the testis.

This LH stimulates the production of testosterone by the testes in males. Thus HCG sends the same message as LH to the testes and results in increased testosterone production by the testes due to HCG's effect on the leydig cells of the testes. In males, hCG mimics LH and helps restore and maintain testosterone production in the testes. If hCG is used for too long and in too high a dose, the resulting rise in natural testosterone will eventually inhibit its own production via negative feedback on the hypothalamus and pituitary.

HCG therapy uses the body's own biochemical stimulating mechanisms to increase plasma testosterone level during HCG therapy. It is used to stimulate the testes of men who are hypogonadal or lack sufficient testosterone.



 
 
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